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monogenic disease

ICD-10 Codes

Related ICD-10:

E20.0 E74.04 M33.02 E71.42 N03.A Q87.41 D57 Q00.1 M87.021 M87.062 E79.81 E83.0 G90.B A50.2 D81.7 Q66.1 E71.121 G72.3 Q39.5 E13.41 Z36.8A G40.A19 M35.0C E71.1 P55.9 G40.83 D57.432 Q85.83 D55.1 G57.93 H15.05 E75.02 M94.1 D57.418 E71.110 Q82.4 N15.0 D82.4 G40.B N01.7 Q82.1 Q24.9 Q20.2 E74.01 G71.3 E71.540 E20.1 M89.73 E80.21 E72.52 G40.A G40.C0 Q22.1 Z22.350 H49.81 G71.13 Q50.39 E70.8 G31.82 E27.1 Q16.3 E05.21 Q64.73 D68.023 Q77.5 E75.00 Q23.1 E72.50 D46.C D56.3 E72.01 E72.02 D84.9 G40.843 Q26.1 N46.125 M30.2 Q87.86 Q61.4 L30.2 D57.219 G40.C Q81.2 G12.24 M41.116 M34.1 D57.09 Q12.0 Q77.3 D57.00 Q71.812 D69.41 E72.22 E74.02 M94.351 N01.8 P09.4 D58.0 Q71.819 Q51.7 H50.812 M61.1 D47.Z2 E71.521 D59.5 E23.2 K20.0 E75.19 E85.82 Q25.44 Q85.82 E10.36 E71.120 M94.352 N06.6 H18.55 D68.52 E88.42 E75.21 D56.5 E75.28 Q37.8 Q64.12 Q95.5 E72.03 D47.Z1 D57.40 E76.22 Q71.0 Q77.6 M25.05 N00.0 E75.26 E83.11 E83.110 G71.02 E74.12 N07.9 N46.01 D72.0 H18.553 E75.09 H50.311 M99.4 F84.2 G12.9 Q24 M85.069 Q75.4 D64.1 Q45.3 E20 G31.81 Q71.81 Q93.51 E75.240 G70.81 Q67 G81.1 A50.42 E72.59 Q23.0 Q87.81 Q87.83 E72.12 Z84.81 G23.3 H80.1 E70.1 E71.41 Q93 A39.81 D61.3 D89.44 G23.1 I69.252 Q71.1 H15.053 M10.09 E71.51 E71.518 G40.834 G71.228 M41.3 D81.4 J84.02 E75.3 Q26.2 H18.52 Q91.7 Z14.0 M89.721 E84 H81.0 G80.1 O45.01 E83.32 J84.81 D61.8 E74.810 G90.1 A50.04 Q89.3 E80.2 G40.C01 Q93.4 E74.819 P78.84 Q85.81 Q79.60 E10.11 M85.071 Q80.4 N07.1 E75.4 E70.311 D89.82 L40.54 B46.4 D80.7 Q62.7 Q13.81 Q13.4 Q07.00 G93.7 D68.022 B42.1 Q72.31 E73.0 E51.2 D57.439 N01.5 G70.80 N07.4 Q32.0 E71.314 E74.29 E34.328 D81.30 H31.29 D55.0 M06.1 E25.9 E72.11 E72.53 E75.25 P94.1 E71.0 Q20.1 Q60.6 I37.0 Q27.8 H31.23 G71.11 G51.39 Q96.0 M61.251 Q77.1 M41.25 G40.812 Q27.4 E80.3 E72.4 C22.2 L10.3 E88.43 G57.90 L40.1 K50.119 M35.03 Q06.0 E80.1 Q91.6 Q71.813 O14.24 M87.065 E75.27 Q96.4 M89.541 E75.243 D57.414 E88.49 E08.10 E75.01 M85.04 Q41.2 K50.10 E88.02 L10.0 F78.A H01.142 G23.0 G24.1 E78.71 E72.23 Q87.85 E71.54 Q44.3 E71.311 G81.02 Q14.1 E78.3 G40.42 Q21.3 A48.4 N03.5 Q82.3 M35.06 N02.1 E75.241 G71.09 C93.00 K90.81 C88.0 N01.1 D57.454 G31.86 Q65.82 H26.043 E88.01 E71.529 E71.11 Q22.0 Q04.1 M08.3 D57.459 I74.11 L87.2 Q22.6 E70.30 E71.440 N01.A K50.8 N07.A E10.3 I45.81 Q56.3 E25.8 E27.40 Q55.3 H30.12 C92.12 E71.52 H31.2 E10 D57.413 Q24.4 Q71.811 Q72.03 D81.818 C93.0 E70.319 Q93.3 D57.42 Z31.44 D68.1 E71.510 E72.21 G37.81 D81.81 Q25.41 Q87.4 E20.818 H47.22 Q74.3 A50.53 E71.520 E76.03 K50.813 D68.029 G12.25 L40.2 A81.1 E80.0 D57.44 E74.00 Q55.7 Q80 M41.112 E70.33 E70.330 E76.02 Q05.9 Q81 Q81.0 D81.6 N01.3 N06.5 E72.1 T88.3 C81.43 Q26.0 M61.29 B38.4 H81.03 Q00.0 E88.40 Q64.70 E85.2 Q81.1 G71.1 G71.12 E75.242 D57.41 Q79.1 E71.542 E71.312 K50.0 E71.511 N17.1 Q91.3 Q37.2 G71.22 L90.1 H31.21 M85.032 E72.51 K50.1 E10.10 Q15.0 C92.A D81.32 E74.82 C93.1 E70.321 Q91.1 Q93.8 Q93.81 Q72.0 C92.20 M05.43 E76.0 E76.01 Q12.3 G56.90 N02.B1 Z15.01 Q77.0 E71.12 H18.5 Q97.0 Q72.02 Q90.1 G40.844 E74.05 E27.0 E74.829 I82.0 K50.11 G40.842 Q91.2 D82.0 D82.1 P09.3 Q64.2 E00.0 H26.063 H80.0 Q52.2 A50.02 H80.12 G51.33 A81.82 R82.992 H47.231 Q81.8 E20.812 I42.1 H18.50 M61.19 E10.A1 D75.84 N02.7 Q72.81 Q72.01 K50.013 R80.3 G40.813 D57.458 E70.318 E72.19 D83 Q43.1 M94.3 I78.0 E72.04 M89.54 D64.3 Q60.1 D80.0 D56.0 E70.0 Z13.79 E79.89 D69.51 Q60.0 E74.20 H26.06 Q20.4 L95.0 P94.0 Q24.6 D59.32 Q87.82 M86.0 M86.461 D70.0 D61.03 E83.59 G51.2 E70.320 E74.11 G47.35 J84.83 E70.328 G62.8 Q31.5 Q93.0 E71.313 Q85.02 E71.111 E70.310 D68.021 H47.03 D57.419 D61.02 D57.45 D57.451 I27.83 Q62.31 M86.369 Q85.03 Q87.42 Q66.0 T80.311 Q67.4 E70.41 Q25.72 E79.1 R82.993 G71.220 L51.3 M61.159 C93.3 C93.32 D57.218 F95.2 J84.841 M12.14 M12.15 Q40.0 G93.42 G71.0342 E88.82 G11.6 L87.0 Q79.4 E76.1 O14.2 P56.90 Q91.0 E88.41 E71.4 E83.119 N05.5 D56.8 A50.56 G11.3 G40.B09 H44.2D3 G71.2 Q75.049 Q77.4 G71.19 D57.433 H01.145 G71.01 Q87.43 G40.C19 Q22.5 E72 E10.4 G40.833 H20.82 N07 D56.1 Q79.63 E70.81 Q93.52 A81.0 G11.1 E10.62 Q78.4 E84.8 G12 Q21.15 Q78.0 H53.63 A50.3 M04.2 M12.1 Q98.1 E75.23 E79.82 H15.052 Q07.01 E77 D57.2 D58 G70.0 H18.54 H18.543 E74.10 M89.52 Q96 D59.39 E10.6 E10.61 E26.02 Q61.1 E25.0 E70.331 G40.84 G40.841 D81.82 H53.51 Q86.1 G61.0 Q04.2 G12.21 Q00.2 Q21.14 E22.9 H47.20 E75.249 Q44.71 H26.03 O26.4 E74.19 E75.1 E75.11 I67.850 M61.173 G56 K31.1 E10.21 E76.210 M93.1 M89.162 D67 H31.20 Q64.8 Q93.2 M31 P91.82 P29.3 E70.32 E70.49 Q78.2 A50.55 G58.8 H35.52 Q87.84 D68.01 E26.81 H50.81 C86.3 D66 E07.0 G12.0 E10.351 M25.0 D69.4 Q60.4 D89 G37.4 M61.2 Q54.4 E20.810 H36.82 D81.3 D81.819 E78.72 N06.21

Description

A monogenic disease, also known as a single-gene disorder, is a diseased condition determined by the interaction of a single gene [1]. This is in contrast to polygenic conditions, where several genes are involved. Monogenic diseases result from single-gene mutations that disrupt the normal functioning of the gene's protein product.

Monogenic diseases can be inherited in an autosomal dominant, autosomal recessive, or X-linked pattern, depending on the location and nature of the mutation [7]. They are relatively rare compared to human diseases that can be categorized as multifactorial and complex.

Examples of monogenic disorders include sickle cell disease, cystic fibrosis, polycystic kidney disease, and Tay-Sachs disease. These conditions are caused by alterations in a single gene and can have significant impacts on an individual's health [15].

Monogenic diseases often exhibit a clear-cut inheritance pattern, making them useful models for studying the genetic basis of complex diseases [2]. For instance, Van der Woude syndrome, which is characterized by lower lip pits and orofacial clefts, has been used as a model for studying the genetics of complex diseases.

Monogenic liver diseases are another area where these conditions play a significant role. These disorders can be caused by mutations in genes involved in liver function, such as those related to metabolism and detoxification [3].

In summary, monogenic diseases are a type of genetic disorder that is determined by the interaction of a single gene. They can have significant impacts on an individual's health and are relatively rare compared to complex diseases.

References: [1] Monogenic Disease Definition. [7] Monogenic inherited diseases are caused by mutation or alteration in the DNA sequence of a single gene. [15] Monogenic diseases are caused by alterations in a single gene. Examples of monogenic disorders are sickle cell disease, cystic fibrosis, polycystic kidney disease, and Tay–Sachs disease.

Additional Characteristics

  • Tay-Sachs disease
  • monogenic disease
  • single-gene disorder
  • autosomal dominant
  • autosomal recessive
  • X-linked pattern
  • sickle cell disease
  • cystic fibrosis
  • polycystic kidney disease
  • Van der Woude syndrome
  • monogenic liver diseases

Signs and Symptoms

Monogenic diseases, also known as single-gene disorders, are caused by mutations in a single gene. The signs and symptoms of these diseases can vary widely depending on the specific gene involved and the individual affected.

Common Signs and Symptoms:

  • Eating and digestive issues, such as difficulty swallowing or an inability to process nutrients [3]
  • Limb or facial anomalies, which include missing fingers or a cleft lip and palate [3]
  • Movement disorders due to muscle stiffness or weakness [3]
  • Neurological issues such as seizures or stroke [3]
  • Poor growth or short stature [3]

Additional Symptoms:

  • Elevated inflammatory markers, fever, leucocytosis, abdominal pain, migrating myalgia, arthralgia, chest pain, and other signs and symptoms [9]

It's worth noting that the specific symptoms of a monogenic disease can depend on the gene involved and the individual affected. Some monogenic diseases may have more severe or complex symptoms than others.

References:

  • [3] Lists various physical anomalies associated with monogenic disorders.
  • [9] Describes additional signs and symptoms associated with certain monogenic conditions.

Please note that these are general symptoms and not all monogenic diseases will present with the same set of symptoms. The specific symptoms can vary widely depending on the disease and individual affected.

Additional Symptoms

Diagnostic Tests

Monogenic diseases, also known as single-gene disorders, are caused by mutations in a single gene and can be inherited from one's parents. Diagnostic tests for these diseases have evolved significantly over the years, offering more accurate and earlier detection.

Traditional Genetic Testing

Traditional genetic testing involves analyzing DNA samples to identify specific genetic variants associated with monogenic diseases [8]. This method is often used when there is a known family history of the disease or when a person has already been diagnosed with it. However, this approach can be limited in its ability to detect all possible mutations and may not provide a definitive diagnosis.

Preimplantation Genetic Testing (PGT)

Preimplantation genetic testing (PGT) is a more advanced diagnostic tool that involves analyzing embryos created through in vitro fertilization (IVF) for specific genetic variants [1]. PGT can be used to detect monogenic diseases, chromosomal structural rearrangements, and aneuploidy. This method provides a definitive diagnosis before implantation, allowing couples to make informed decisions about their reproductive choices.

Next-Generation Sequencing (NGS)

Next-generation sequencing (NGS) is a powerful diagnostic tool that enables the simultaneous analysis of multiple genes and genetic variants [2]. NGS can be used to detect monogenic diseases, including those caused by maternally inherited mutations or X-linked recessive disorders. This method provides a comprehensive understanding of an individual's genetic makeup.

Full-Genome Analysis (FGA)

Full-genome analysis (FGA) is another advanced diagnostic tool that combines long-range assembly and whole-genome sequencing to identify genetic variants associated with monogenic diseases [3]. FGA can provide a more accurate diagnosis than traditional genetic testing methods, especially in cases where there is no known family history of the disease.

Non-Invasive Prenatal Diagnosis (NIPD)

Non-invasive prenatal diagnosis (NIPD) involves analyzing cell-free DNA from maternal blood to detect fetal genetic variants associated with monogenic diseases [11]. NIPD can be used to determine fetal sex, RHD status, and some monogenic diseases. This method is particularly useful for pregnancies at risk due to a positive family history or abnormal sonographic findings.

Quest Diagnostics Difference

Quest Diagnostics offers advanced diagnostic testing services that combine quality, speed, and technology to power accurate diagnostics [10]. Their services include genetic testing for monogenic diseases, among other conditions. Quest's commitment to providing convenient and affordable diagnostic testing options makes it an attractive choice for individuals seeking accurate diagnoses.

References:

[1] Preimplantation genetic testing (PGT) can be performed for monogenic disorders or single gene defects (PGT-M), for chromosomal structural rearrangements (PGT-SR), and for aneuploidy detection (PGT-A).

[2] Next-generation sequencing (NGS) makes it possible to test for maternally inherited mutations and/or X-linked recessive disorders.

[3] Full-genome analysis (FGA) combines long-range assembly and whole-genome sequencing to identify genetic variants associated with monogenic diseases.

[8] Traditional genetic testing involves analyzing DNA samples to identify specific genetic variants associated with monogenic diseases.

[10] Quest Diagnostics offers advanced diagnostic testing services that combine quality, speed, and technology to power accurate diagnostics.

[11] Non-invasive prenatal diagnosis (NIPD) involves analyzing cell-free DNA from maternal blood to detect fetal genetic variants associated with monogenic diseases.

Additional Diagnostic Tests

  • Traditional Genetic Testing
  • Preimplantation Genetic Testing (PGT)
  • Next-Generation Sequencing (NGS)
  • Full-Genome Analysis (FGA)
  • Non-Invasive Prenatal Diagnosis (NIPD)

Treatment

Treatment Options for Monogenic Diseases

Monogenic diseases, also known as single-gene disorders, are caused by mutations in a single gene and can be severe, life-long, and lethal. While there is no cure for these diseases, various treatment options have been developed to manage their symptoms and improve quality of life.

Small-Molecule Drugs

Small-molecule drugs are still the predominant treatment strategy for most monogenic disorders [6]. These medications target specific enzymes or proteins involved in the disease process and can help alleviate symptoms. However, they generally do not address the underlying genetic mutation causing the disease.

Gene Therapy Approaches

New gene therapy approaches have emerged as promising treatment options for rare congenital disorders and certain tumor entities [1]. Gene therapy involves introducing a healthy copy of the faulty gene into the patient's cells to restore normal protein production. This approach has shown significant promise in treating monogenic diseases, particularly those caused by mutations in genes involved in protein production.

Approved Treatments

Several treatments have been approved for use in patients with monogenic diseases [2-5]. These include:

  • Luxturna (RPE65-mutation-associated retinal dystrophy)
  • Zolgensma (spinal muscular atrophy)
  • Strimvelis (adrenoleukodystrophy)
  • Lyfgenia (Fabry disease)

RNA-Based Approaches

RNA-based approaches have also been developed to treat monogenic diseases [9]. These methods involve using RNA molecules to target specific genes or proteins involved in the disease process. This approach has shown promise in treating various monogenic disorders, including those caused by mutations in genes involved in protein production.

Challenges and Future Directions

While significant progress has been made in developing treatment options for monogenic diseases, several challenges remain [4]. These include the need to improve delivery methods, increase efficacy, and reduce toxicity. Researchers are also exploring new strategies to overcome these challenges and develop more effective treatments for these devastating disorders.

References:

[1] Kirschner, J. (2020). New gene therapy approaches for rare congenital disorders and certain tumor entities. [Context result 1]

[2] Baylot, V. (2024). Four treatments are now approved for monogenic diseases. [Context result 2]

[3] Clarke, LA. (2023). Nusinersen: A treatment for spinal muscular atrophy. [Context result 3]

[4] Zhang, Y. (2023). Gene therapy and delivery tools for monogenic diseases. [Context result 4]

[5] (2020). Gene therapy: A natural fit for solving monogenic diseases like SMA. [Context result 5]

[6] Wang, Y. (2021). Small-molecule drugs in treatment strategies for rare monogenic disorders. [Context result 6]

[7] Brooks, PJ. (2023). FDA-approved products using AAV for gene therapy to treat monogenic diseases. [Context result 7]

[8] (2020). Gene therapy approved for patients without a suitable donor for bone marrow transplantation. [Context result 8]

[9] Clarke, LA. (2023). RNA-based approaches to treat monogenic diseases. [Context result 9]

[10] Monogenic diseases: A review of the current understanding and treatment options. [Context result 10]

Recommended Medications

  • Luxturna
  • Zolgensma
  • Strimvelis
  • Lyfgenia

💊 Drug information is sourced from ChEBI (Chemical Entities of Biological Interest) database. Always consult with a healthcare professional before starting any medication. Click on any medication name for detailed information.

Differential Diagnosis

The differential diagnosis of monogenic diseases involves identifying and distinguishing between various genetic disorders that can present with similar symptoms or characteristics.

According to [1], the differential diagnosis of various types and forms of diabetes is of great practical importance, particularly for monogenic diseases. This is because monogenic diseases are caused by a single gene mutation, which can lead to distinct clinical features and diagnostic challenges.

Some examples of monogenic diseases that require differential diagnosis include:

  • Fragile X syndrome (FXS) [3]
  • PTEN hamartoma tumor syndrome (PHTS) [3]
  • Tuberous sclerosis (TSC) [3]
  • Rett syndrome (RTT) [3]

In addition, the differential diagnosis of monogenic diseases may also involve distinguishing between different types of diabetes, such as:

  • Maturity-onset diabetes of the young (MODY) [10]
  • Congenital (neonatal) diabetes [10]
  • Syndromic types of diabetes [10]

The differential diagnosis of monogenic diseases can be complex and requires a thorough understanding of the underlying genetic mechanisms and clinical features. It is essential to consider multiple factors, including family history, age of onset, and presence of associated manifestations, when making a differential diagnosis.

According to [12], the differential diagnosis between ADPKD and other cystic kidney diseases depends on the age of the patient, family history, and the presence of associated manifestations. Similarly, the differential diagnosis of monogenic diabetes from other forms of diabetes requires careful consideration of clinical features and genetic testing results.

In some cases, the absence of islet-autoantibodies may be a useful indicator in distinguishing between monogenic autoimmune diseases, such as autoimmune polyendocrinopathy syndrome type 1 (APS1) [13].

Overall, the differential diagnosis of monogenic diseases requires a comprehensive approach that takes into account multiple factors and clinical features.

References:

[1] Owen KR. The differential diagnosis of various types and forms of diabetes. 2009. [3] Tinker RJ. Diagnostic delay in monogenic disease is reportedly common. 2024. [10] Babcock SJ. Monogenic intestinal epithelial disorders, also known

Additional Information

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