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AGAT deficiency

ICD-10 Codes

Related ICD-10:

D70.4 H18.233 I82.511 E61.3 E80.5 M61.262 D61.0 E75.240 G70.81 M85.46 E72.59 H60.8X3 E70 Q87.81 E50.2 T56.5 Q71.3 D68 E71.41 M12.131 H18.043 E71.310 E71.518 P74.42 M08.89 D81.4 K05.22 K05.222 M11.062 H35.44 K11.2 N04.6 E72.20 G40.801 Q37.5 E74.820 M11.011 M89.72 N25 P28.41 H81.0 O45.019 D81.39 E83.32 E74.810 E00.1 E72.04 H35.17 H18.02 D64.3 M76.811 H35.31 M62.52 K31.81 E80 E70.0 E79.8 H18.831 E13.5 P94.0 N02.B5 E88.09 D70.0 G51.2 G71.032 P70 E74.11 E80.20 A66.5 M92.1 G43.60 E70.29 E70.328 M60.11 P59.1 Q43 E71.313 N07.3 E71.111 M11.072 H35.023 D61.01 G73.1 D68.021 E74.03 H35.22 M86.369 P76.0 Q87.11 E70.41 Q25.72 E70.21 E79.1 G71.220 G40.C09 E22.2 M12.15 E88.811 M87.872 E00.2 D56.2 M86.311 C93.30 D68.023 E75.00 P93.0 G61.9 M61.252 M02.16 E83.31 M86.10 D81.5 E27.8 E72.02 P61.3 M86.09 Q87.86 J84.03 N52.33 G44.031 P61.1 G40.C Q81.2 M89.042 E42 E45 M86.29 E74.9 M89.021 M12.18 K51.219 E72.22 D81.1 Q51.7 N13.6 E80.2 E72.29 P74.6 M11.022 O45.011 M86.321 N02.B4 G81.00 Q80.4 D80.2 N07.1 E70.311 M86.331 D80.7 G93.7 H53.69 N46.025 E79.2 E73.0 M85.121 E74.29 D81.30 O35.19 T86.85 Q55.4 E72.53 E75.25 E71.0 M60.131 Q60.6 M86.039 G51.39 M86.39 M61.251 I73.81 T44.0X1 E72.4 M31.31 M89.529 M87.837 D84.0 E80.1 G40.B01 E83.2 N31.2 M87.065 E75.27 M12.132 E75.243 G37.9 E88.49 M86.312 Q82.0 M86.28 E75.01 P56.0 E76.1 H02.432 N03 E71.4 E64.3 J84.842 N36.42 Q78.5 G93.49 E70.339 Q45.8 E75.248 G40.C19 E72 E72.2 M11.03 G44.099 M89.53 M61.20 T39.316 M86.551 Q41.2 M12.05 E75.0 P09.8 M11.051 M11.052 E75.22 Q39.4 M61.232 E78.71 K11.0 Q39.8 E51.11 H35.173 P57 P57.0 M1A.452 E71.311 G40.42 N02.1 E75.241 Q79.6 M61.579 H26.043 E88.01 M10.45 M61.261 M11.0 G71.034 J34.81 M61.452 L87.2 T80.310 E71.440 N01.A B46 N25.9 M12.35 M25.011 M86.511 H81.01 O46.013 D69.6 E80.6 N13.9 Q55.3 L74.52 E71.52 E31.8 H04.211 E70.319 H10.511 G60.1 M87.038 E71.510 E72.21 D81.81 I77.8 E71.520 G12.25 L40.2 M86.332 E80.0 Q80 Q80.1 M11.02 E70.33 E76.02 G04.30 K55.21 L66 E70.329 E34.8 H60.20 M12.13 L90.3 M87.04 E71.43 Q81.1 N07 P77 M12.141 E70.81 N02.B N03.7 Q93.52 B46.1 Z83.42 M12.1 E75.2 E75.23 E79.82 H15.052 D81.31 I63.32 I82.54 M12.171 D61 D72.8 Q61.1 E25.0 E70.331 H05.82 K76 N02.4 C94.2 A08.11 Q25.4 E75.249 J84.82 G04.32 E75.11 I67.850 Q61.00 M61.24 R60 R74 E76.211 Q79.5 N14.11 M86.14 G51.32 K08.26 N91.0 O92.79 M89.731 E71.528 E10.A P61.5 K05.221 G04.3 M11.08 P09.2 G11.11 G12.0 H80.2 M89.031 M61.2 M61.469 Q54.4 A50.51 D81.3 D81.819 E78.72 M12.149 E20.811 E71.521 D59.5 E71.120 M94.352 E88.42 E75.21 O45.00 E85.1 K85 E75.28 H26.033 D75.839 Q64.12 R26.8 E72.03 M11.012 D57.4 E76.22 Q77.6 A39.1 P27.0 E75.26 M95 E74.12 N46.01 M61.211 D81.810 E75.09 M12.19 F84.2 E71.32 D64.1 E70.89 G71.12 N18.5 E75.242 E85.0 M87.80 R77.0 E72.81 E71.511 N17.1 R26.0 G11.5 L81.6 G71.22 M12.179 E72.51 I69.393 H92.21 D81.32 E70.321 E72.89 E76.01 E83.39 E74.818 E70.2 D73.81 M48.17 E75.244 G90.3 E83.81 E74.05 E74.829 Q80.3 I27.1 G83.5 P09.3 Z14.02 Q07.8 E00 E74.2 N04.5 L95.1 A81.82 A36.84 T86.8 H60.2 E79.9 I63.333 E61.0 R80.3 G40.813 E72.19 M60.132 M86.34 E74.04 H31.123 M1A.351 O91.23 M11.07 G90.523 P74.5 K51.312 E79.81 J41.1 G90.B N07.5 M89.041 D81.7 E72.09 I69.86 M86.16 H26.031 L44.4 I69.212 E71.1 O72.3 H26.041 G40.83 H35.021 M12.151 E75.02 P96.82 E71.110 N15.0 G43.609 P71.1 P78.8 H16.051 K05.213 E83.09 E74.01 M87.051 E80.21 M11.032 E72.52 H49.81 E74.31 E70.8 G31.82 M10.451 E72.8

Description

AGAT Deficiency Description

AGAT deficiency, also known as Arginine:glycine amidinotransferase deficiency, is an inherited disorder that primarily affects the brain [2]. It is caused by changes in the GATM gene and is inherited in an autosomal recessive manner [4].

Individuals with AGAT deficiency generally have mild to moderate intellectual disability [4][8]. Other signs and symptoms may include seizures, delayed speech development, delayed motor skills development, such as sitting and walking [8], and muscle weakness [2].

AGAT deficiency is a treatable condition that primarily affects the brain. Early diagnosis may prevent IDD (Intellectual Disability Disorder) and myopathy [5]. Patients with unexplained IDD with and without muscle weakness should be tested for AGAT deficiency.

The creatine biosynthesis disorders, including AGAT deficiency, comprise three disorders: guanidinoacetate methyltransferase (GAMT) deficiency, L-arginine:glycine amidinotransferase (AGAT) deficiency; and creatine transporter (CRTR) deficiency [13]. Developmental delay and cognitive dysfunction or intellectual disability and muscle weakness are characteristic features of these disorders.

References: [2] - Arginine: glycine amidinotransferase deficiency is an inherited disorder that primarily affects the brain. [4] - AGAT deficiency is caused by changes in the GATM gene and is inherited in an autosomal recessive manner. [8] - Mild to moderate intellectual disabilities, delayed speech development, delayed motor skills development, such as sitting and walking [13] - The creatine biosynthesis disorders, including AGAT deficiency, comprise three disorders: guanidinoacetate methyltransferase (GAMT) deficiency, L-arginine:glycine amidinotransferase (AGAT) deficiency; and creatine transporter (CRTR) deficiency.

Additional Characteristics

  • muscle weakness
  • seizures
  • mild to moderate intellectual disability
  • delayed speech development
  • delayed motor skills development, such as sitting and walking

Signs and Symptoms

AGAT Deficiency Signs and Symptoms

Individuals with Arginine:Glycine Amidinotransferase (AGAT) deficiency often experience a range of symptoms, which can vary in severity and impact different aspects of their lives. Here are some common signs and symptoms associated with AGAT deficiency:

  • Intellectual Disability: People with AGAT deficiency may have mild to moderate intellectual disabilities, affecting their cognitive development and ability to learn [4].
  • Delayed Speech Development: Delayed speech development is a characteristic feature of AGAT deficiency, which can impact communication skills and language abilities [2][5].
  • Muscle Weakness: Muscle weakness is another common symptom of AGAT deficiency, which can affect motor skills development and overall physical strength [5].
  • Global Developmental Delay: Infants with AGAT deficiency may experience global developmental delay, which can manifest as delayed milestones in various areas, such as sitting, walking, or talking [2].
  • Autistic-like Behavior: Some individuals with AGAT deficiency may exhibit autistic-like behavior, including social interaction difficulties and repetitive behaviors [8].

These symptoms can have a significant impact on daily life, making it essential for early diagnosis and intervention to manage the condition effectively.

Additional Symptoms

  • Intellectual Disability
  • Global Developmental Delay
  • Delayed Speech Development
  • Autistic-like Behavior
  • muscle weakness

Diagnostic Tests

Diagnostic Tests for Arginine: Glycine Amidinotransferase (AGAT) Deficiency

To diagnose AGAT deficiency, several tests can be performed to assess the levels of certain metabolites in the urine and plasma. Here are some diagnostic tests that may be used:

  • Urine and serum/plasma testing: This is an initial test to rule out creatine deficiency syndromes following clinical presentation [3]. Low plasma and urinary creatinine values are a diagnostic clue for AGAT and GAMT deficiency [6].
  • Determination of urine/plasma GAA levels: Patients with unexplained IDD with and without myopathy should be assessed for AGAT deficiency by determination of urine/plasma GAA levels [2].
  • Cerebral creatine levels: Determination of cerebral creatine levels can also help diagnose AGAT deficiency [1].

It's worth noting that testing sensitivity may increase if both plasma and urine sample types are used [8]. Additionally, genetic counseling is recommended for conditions related to AGAT deficiency [4].

References:

  • [1] Arginine: glycine amidinotransferase deficiency (AGAT) is one of the three cerebral creatine deficiency syndromes (CCDS).
  • [2] Patients with unexplained IDD with and without myopathy should be assessed for AGAT deficiency by determination of urine/plasma GAA levels.
  • [3] Initial tests to diagnose or rule out creatine deficiency syndromes following clinical presentation; Urine and serum/plasma testing should be ...
  • [4] This gene encodes AGAT, which converts arginine and glycine to ornithine and guanidinoacetate in the creatine cycle pathway. Genetic counseling. AGAT deficiency ...
  • [6] Low plasma and urinary creatinine values are a diagnostic clue for AGAT and GAMT deficiency.
  • [8] For the diagnosis of AGAT or GAMT deficiency, either plasma or urine can be used, although testing sensitivity may increase if both sample types ...

Additional Diagnostic Tests

  • plasma testing
  • plasma GAA levels
  • Cerebral creatine levels

Treatment

Treatment Options for Arginine: Glycine Amidinotransferase (AGAT) Deficiency

Individuals with AGAT deficiency often benefit from early treatment with creatine supplementation, which can significantly improve their condition.

  • Creatine Supplementation: Studies have shown that oral creatine monohydrate supplementation, in combination with ornithine supplementation, can lead to significant improvement in patients with AGAT and GAMT deficiencies [2][3]. A dose of 400 mg/kg per day has been employed, resulting in notable improvements [6].
  • Early Intervention: Early treatment is crucial in preventing or minimizing psychomotor delay and other complications associated with AGAT deficiency. If creatine supplementation is administered early enough, seizures and failure to thrive may also be avoided [4].

Treatment Consists Of

Oral high-dose creatine monohydrate, combined with ornithine supplementation (100-800 mg/kg/day given in 3-6 doses daily) and/or other supportive therapies, has been found to be effective in managing AGAT deficiency symptoms.

  • Creatine Monohydrate: This is the primary treatment for AGAT deficiency, which helps restore normal creatine levels in the body.
  • Ornithine Supplementation: Ornithine is an amino acid that plays a crucial role in the synthesis of guanidinoacetate. Supplementing with ornithine can help alleviate symptoms associated with AGAT deficiency.

References

[1] S Stockler-Ipsiroglu · 2015 · Cited by 64 [2] G Fernandes-Pires · 2022 · Cited by 33 [3] JF Clark · 2015 · Cited by 61 [4] Ozempic Could Help Curb Alcoholism. [5] Drug use disorders are associated with significant costs to society due to lost productivity, premature mortality, increased health care expenditure, and costs related to criminal justice, social welfare, and other social consequences.

Recommended Medications

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Differential Diagnosis

Understanding Differential Diagnosis in AGAT Deficiency

The differential diagnosis for AGAT (Arginine:Glycine Amidinotransferase) deficiency involves considering other conditions that may present with similar symptoms. In the case of AGAT deficiency, a partially treatable condition, it is essential to rule out other genetic myopathies and cerebral creatine deficiencies.

Conditions to Consider in Differential Diagnosis

  • L-Arginine:Glycine Amidinotransferase (AGAT) Deficiency: This is another creatine biosynthesis disorder that can present with similar symptoms, including developmental delay and cognitive dysfunction.
  • X-linked Creatine Transporter (CRTR) Deficiency: This condition affects the transport of creatine in the brain and can also present with similar symptoms.

Diagnostic Tools

To confirm a diagnosis of AGAT deficiency, several diagnostic tools can be used:

  • Brain Magnetic Resonance Spectroscopy (MRS): This non-invasive test can help identify abnormalities in brain chemistry that may indicate AGAT deficiency.
  • Guanidinoacetate and Creatine Determination: Measuring the levels of guanidinoacetate and creatine in urine and plasma can also help confirm a diagnosis of AGAT deficiency.

Importance of Accurate Diagnosis

Accurate diagnosis is crucial for providing appropriate treatment and management for individuals with AGAT deficiency. By considering other conditions that may present with similar symptoms, healthcare providers can ensure that patients receive the best possible care.

References

  • [3] AGAT deficiency is an autosomal recessive cerebral creatine deficiency caused by a deficiency of the enzyme arginine:glycine amidinotransferase.
  • [4] The differential diagnosis in children with a cerebral creatine deficiency includes L-Arginine:glycine amidinotransferase (AGAT) deficiency and X-linked ...
  • [12] If a single pathogenic variant or variants of unknown significance in GATM are identified, AGAT activity can be measured in fibroblasts 8 to confirm the diagnosis of AGAT deficiency.

Additional Differential Diagnoses

Additional Information

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disease_ontology
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lschriml
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2012-05-22T11:53:57Z
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DOID:0050712
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MIM:612718
IAO_0000115
An amino acid metabolic disorder that has_material_basis_in a mutation in the GATM gene resulting in deficiency of arginine:glycine amidinotransferase which then limits creatine synthesis.
oboInOwl#hasExactSynonym
Cerebral creatine deficiency syndrome 3
IDO_0000664
http://purl.obolibrary.org/obo/GENO_0000148
relatedICD
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AGAT deficiency
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https://w3id.org/def/predibionto#has_symptom_749
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